schistosomiasis

created by craigc
(thing) by Excalibur (2.2 d) (print)   (I like it!) 4 C!s Mon Aug 22 2005 at 8:15:45

Also see Humanopathogenic worms

Once again, I find myself writing about that grotesque but nonetheless compelling topic - parasitology! If for some reason you don't want to read about tiny little critters burrowing through your skin and growing up into worms that release thousands of eggs into your body, then turn back now. Otherwise, continue to learn about one of the world's most pressing public health issues.


The basics

Schistosomiasis, or bilharzia, is a disease caused by various flatworms of genus Schistosoma, contracted when tiny larvae burrow into your skin when you step into contaminated water. That's right, folks - it's not enough to avoid eating when in certain parts of the world. Horrible things can happen to you just as a result of bathing or fetching water from a river. Worldwide, there are around 200 million people infected; the World Health Organization and many countries are working to eradicate this disease. Nevertheless, it remains common throughout the tropics, in the Caribbean, eastern South America, Africa, the Middle East, and Southeast Asia - 74 countries in total. In fact, it's so common in certain communities in Africa that infection with it is treated as a rite of passage.

Schistosomes belong to phylum Platyhelminthes, class Trematoda. Trematodes, also called flukes, are rhomboid in shape and responsible for many parasitic infections in humans. Schistosomes, or blood flukes, cause a chronic disease that damages the urinary tract, bowel, and liver in infected individuals. The disease is rarely fatal but can be extremely painful. Five main species affect humans: S. mansoni, present in Africa, the Middle East, and Brazil; S. haematobium, in Africa and the Middle East; S. japonicum, mainly in China; and the slightly rarer S. mekongi (found around the Mekong River) and S. intercalatum (certain parts of West Africa).

The disease was known in ancient Egypt; it is named for Theodor Bilharz, the German pathologist who first discovered the worm responsible. It is most prevalent in women and children; children frequently play in the water and are thus more likely to be infected. Women catch the disease by cleaning and doing laundry in contaminated water sources. Fishermen, not surprisingly, are also frequently infected.

The disease has major social and economic effects in areas where it's endemic. Even if most people have only mild symptoms, it has a major overall effect on economic activities and school performance is hurt in children infected with the worms. The disease is extremely common in some areas - around Lake Volta in Ghana, over 90% of children are infected. Incidence is so high in Egypt that bladder cancer secondary to schistosomiasis infection is the leading cause of death in men between 20 and 44 - an age rarely affected by bladder cancer in the developed world.


Memoirs of a schistosome

Let's start at the beginning. Okay, so someone infected with the disease releases eggs in their feces or urine. In areas with inadequate sanitation (those who've been following my writeups on parasites will note that inadequate sanitation is a major theme here), human waste eventually makes its way into rivers or streams. The eggs, upon reaching water, hatch into little baby miracidia, which then penetrate the schistosome's intermediate host, a snail. (Many parasites have two hosts in their life cycle: a definitive host, where they reproduce, and an intermediate host, where they spend another portion of their life.)

Once inside the snail, the miracidium then moves on to the next stage of its life: it grows up into a sporocyst, which is a sac-like larval stage. After two sporocyst stages, it breaks up into thousands of cercariae, which emerge from the snail in a daily cycle. The cercariae are able to swim, and they respond to turbulence in the water and chemicals detected from human skin. They explore and then attach to any skin they manage to find, and secrete enzymes that break down the skin, permitting them to burrow in.

At this point, the cercariae shed their tails, becoming schistosomulae. There won't be a test; you are not required to memorize all these words. At any rate, they migrate through the circulatory system into the lung, and thence to the liver, where they develop into their adult forms and begin feeding on red blood cells. Adult schistosomes average about a centimeter in length, and they are dioecious - that is, they have two distinct sexes.

Female and male worms pair up, and unlike televangelists they are completely faithful to one another; in fact, they attach to each other and the larger female worms become completely dependent upon males for nutrition. They travel through the circulatory system to their permanent home, whereupon they attach to the walls of veins surrounding the intestines or, in the case of S. haematobium, the bladder.

The happy couple finally starts having babies - they lay eggs that work their way through the intestinal lining or into the ureters and bladder. Adult worm pairs live on average for five years, and they release hundreds or thousands of eggs per day depending on their species. The eggs are released when the human host goes potty, thereupon starting the next generation.


So what's it like to have schistosomiasis?

It's not great, let's face it. It doesn't start out too bad. An itchy rash occasionally occurs within a few days of infection; within a month or two, a cough, fever, chills, and muscle aches can happen. In severe infection, particularly by S. japonicum and S. mansoni, symptoms may be bad enough to severely sicken or kill the patient; this syndrome is called Katayama fever. In most patients, though, this stage is asymptomatic.

After awhile, though, it gets worse. Those eggs we talked about, that burrow through the lining of the intestines and bladder? Over years and years, that causes long term damage. Bleeding is a common symptom - bloody shit, or in the case of the bladder-dwelling S. haematobium, bloody piss, is common in affected people. The intestinal wall and bladder can become scarred as time passes. Occasional bouts of bloody diarrhea affect those with intestinal varieties, seriously impacting their success in the dating world. Abdominal pain, fever, and fatigue occur as well; colon damage can accumulate until a chronic inflammation develops and polyps form; patients are at increased risk of other bowel diseases due to the worms' damage. Bladder cancer is common in people with urinary schistosomiasis, as are other problems with the kidneys and bladder.

Not all the eggs make it. Many are instead swept up in the blood and they end up in the liver. The eggs themselves are harmless, but the body's immune response results in inflammation and eventual damage, as granulomas - fibrous lesions - form around the eggs. An enlarged, diseased liver and spleen are common after long-term infection. Rarely, eggs can make their way to the brain or spinal cord, which can lead to inflammation and seizures or paralysis. Occasionally the skin, lungs, adrenal glands, or eyes can be involved as well. Some varieties cause sores on the vulva that increase the likelihood of HIV transmission.

Even after the initial infection clears, which takes a number of years, reinfection is common, compounding damage to the bladder, intestine, and liver from the previous go-round.


So what can I do if I get it?

The disease is diagnosed by examination of the stool or urine for eggs, which may have to be repeated over time as release of eggs can be intermittent. As it happens, schistosomiasis is eminently treatable. A single dose of praziquantel, a common drug used to treat parasites, will clear up infection. It's a remarkably safe drug; before its invention, therapy was highly dangerous. However, where the disease occurs, reinfection is common and broader efforts are needed to control the spread of the parasite by interfering with other phases of its life cycle.


Well then, what are people doing about it?

The World Health Organization and various other international organizations and NGOs (non-governmental organizations) formed the Partners for Parasite Control in 2001 to help control parasitic infections. One of the organization's goals is to regularly treat 75% of school children infected with the disease by 2010, and to ensure that drugs are available for it in every country where it occurs. Targeting children in particular is reasonable because it's easiest to achieve widespread treatment by using school personnel in the process and because children are more frequently infected than adults. Sadly, the 20¢ average cost to treat a child for schistosomiasis is a serious barrier to adequate control of the disease.

The Partners' current strategy for treatment involves providing praziquantel to every child in regions where the disease is rampant; since reinfection is often inevitable, ensuring that people are and remain completely cured is less useful than simply working continuously to ensure that no child is severely infected. The number of worms present is directly related to the severity of damage; if few are present, the likelihood of symptoms developing is low.

Some areas use other strategies as well as drug treatment. Other steps to control the disease involve the more generally useful step of making sure adequate sanitation exists so that doody doesn't end up contaminating the water supply. Streams and lakes heavily populated with the snails that harbor the intermediate stages of the parasite are treated to kill the snails off, which can very significantly reduce the rate of infection.


Sources
(thing) by vidwan (7.3 mon) (print)   (I like it!) 1 C! Mon Dec 25 2006 at 12:30:39

Surviving lifelong copulation in a hostile milieu

Mammalian blood is among the most hostile habitats for an alien organism. An intricate arsenal of immunological weapons, cellular and humoral, can usually destroy trespassers. A creature that not only dwells in the human blood stream, but incessantly mates in this unfriendly love nest is the blood fluke called schistosoma. The schistosomatic couple affords a romantic sight in copula under the microscope. Genus Schistosoma (Latin: split body) belongs to phylum Platyhelminthes (flat worms), class Trematoda

Global pestilence

This parasite causes schistosomiasis, a major source of morbidity and mortality for developing countries in Africa, South America, the Caribbean, the Middle East, and Asia. It is a chronic disease that damages the urinary tract, intestines and liver in infected individuals. Most human schistosomiasis is caused by the species Schistosoma haematobium, Schistosoma mansoni, or Schistosoma japonicum. Named after the German pathologist in Cairo, Theodor Bilharz, who first described the cause of urinary schistosomiasis, bilharzia, or bilharziosis, is the eponym for schistosomiasis in many countries. At least 200 million people in at least 74 countries have active schistosomal infection. Of these, approximately 120 million have symptoms, and 20 million are severely ill. Disease prevalence tends to be worse in areas with poor sanitation, increased freshwater irrigation usage, and heavy schistosomal infestation of human and/or snail populations.

Romantic worms. Anatomic adaptation

The schistosomes are dioecious, i.e., the sexes are separate and the two sexes display a strong degree of sexual dimorphism, being dissimilar in appearance. Their body structure particularly that of the female, is clearly an adaptation to an intravascular existence. Female flukes are long and slender (1.2 to 2.6 cm in length). The ovary is situated behind the midpoint of the body and the uterus contains 20 to 30 eggs. The male is shorter (from 0.6 to 2.2 cm long) and possesses 4 to 8 testes. While the body is incurved ventrally to form a groove called gynaecophoric canal. The male surrounds the female and embraces her within his gynaecophoric canal for the entire adult lives of the worms in monogamy, reproducing sexually. The anatomy of the slender worms is clearly streamlined to locomotion in blood vessels.

Immune camouflage

In addition to their structural adaptation, nature has provided these parasites immunological strategies to survive in the blood vessels. They are masters at evading their host's immune system, surviving for years in the blood stream. The worms manipulate their surfaces to deceive the host to be accepted by the receptors of the venous endothelium and develop armours ( teguments) that are highly resistant to host immune responses. They steal and incorporate host proteins, including blood group antigens, in their integuments. Coated in host antigens, and apparently unaffected by host immune responses, schistosomes can live comfortably for years in the mammalian blood stream, in some cases, for up to 40 years. As a result, those who live in endemic areas are at constant risk of re-infection, which can be very rapid.
When it facilitates reproduction, the parasites shed their immune mask and trigger inflammatory response of the host tissue. The parasite's continued transmission is actually dependent on their host's immune response. Schistosoma eggs secrete antigens to induce an intense granulomatous response that facilitates their passage from the blood stream to the gut lumen and, thus, to the outside world. Eggs require this intense immune response to aid their migration through the body.

Bloodthirsty parasites

Living in the blood stream, these flukes drink blood. The origin of the word parasite is derived from the respectable Greek word parasitos, meaning quite mundanely a 'fellow-diner' or 'guest' to the house. Biologically, parasitism is defined as a type of symbiosis where two (or more) organisms from different species live in close proximity to one another, in which one member depends on another for its nutrients, protection, and/or other life functions. The dependent member (the parasite) benefits from the relationship while the other one, the host, is harmed by it. ( BioTech Dictionary at http://biotech.icmb.utexas.edu/.)

Power struggle between man and worm

Biological theories attempt to explain sexual reproduction as a source of variability that drives evolutionary processes as a result of organisms attempting to boost their resistance to parasite infection. This struggle for survival between host and parasite, in which both parties evolve as fast as they can in order to live and prosper, has been described as the Red Queen Effect by the late evolutionary biologist William Donald Hamilton, who postulated that sex evolved because new combinations of genes could be presented to parasites - organisms with sex were able to continously run away from parasites. In his book The Red Queen, Matt Ridley expounds on this and interesting aspect of parasitism. Parasites evolve toward greater infectious capacity just as quickly as hosts evolve stronger resistance. Hamilton and Ridley argue that sex has been humanity's most effective weapon for the war on parasites, both internal and external.

Life cycle

Eggs passed in the urine or feaces of an infected person and hatch in fresh water to release larvae, which infect the intermediary host, freshwater snails, which release large numbers of fork-tailed free-swimming larvae called cercariae into water. Here they may find the definitive host, a human wading in water, or die. Cercariae bore through the skin to veins and pass into the lungs before reaching the portal vein, where they metamorphose and mature, shedding their tails. Here, the permanent copulation starts. Together, the male embracing the female in his gynaecophoric canal, they happily swim against the blood current to the veins of the gut or bladder, where the female begins to lay egg, which migrate through the bowel or bladder wall to be shed via feaces or urine. Eggs that are not shed successfully may remain in the tissues or be swept back to the liver or to lungs.
The pathology of chronic schistosomiasis, which is far more common than the acute form of the infection, results from immune response to the eggs, including granuloma formation and fibrotic changes. Schistosomiasis is precancerous. Cancer of the urinary bladder often develops in chronic S haematobium infection.

Treatment

The therapy of choice is with Praziquantel, which destroys the worms by increasing their cell membrane permeability, resulting in loss of intracellular calcium, massive contractions, and paralysis of their musculature, followed by attachment of phagocytes to the parasite and its death. Owing to reinfection, children in endemic areas, should be treated twice a year.

Prevention strategies and prospects

The prevalence of schistosomiasis is increasing world wide. Transmission is usually associated with poor socio-economic conditions. In highly endemic areas children start to accumulate worms as soon as they are old enough to have water contact and may, because of the chronic nature of the infection and continued susceptibility to re-infection, remain infected throughout their lives.
Mass treatment of the population (especially children) is effective in decreasing the egg load in the community. Attempts to control or eradicate the intermediate snail host by mollusciciding chemotherapy have generally been unsuccessful.
The progress made has been more than counterbalanced by increases in the incidence of schistosomiasis that have accompanied social dislocation and mass migrations caused by war, drought and famine, and by the effects of man-made ecological changes, unfortunately including many water resource development projects, with the engineers refusing to listen to epidemiologists, although dams can be made less `snail-friendly'.
The only lasting solution would be global access to a safe water supply, improved sanitation, including the provision of latrines, and health education. The reality is that billions of people lack this luxury. As long as the mankind accepts and neglects poverty, and humans parasitize humans, this vision will remain an illusion. As a species, humans appear to be losing the collective evolutionary race against many other parasites.

Sources

http://www.emedicine.com/emerg/topic857.htm
http://www.tropeduweb.ch/ts/factsheets/d34/fs_d34.htm
http://www.york.ac.uk/res/schisto/background.htm
http://www.path.cam.ac.uk/~schisto/
http://who.int/wormcontrol/en/
http://www.uow.edu.au/arts/sts/bmartin/dissent/documents/AIDS/Hamilton05.pdf
http://www.springerlink.com/content/w5ba1mlgf41g5k8h/


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